The BRAF gene is located on the long (q) arm of chromosome 7 at position 34.

The SHH gene, Sonic the Hedgehog gene, no joke, it's crazy

BRAF Gene Mutation Tests: Reference Range, Interpretation, Collection and Panels

BRAF Gene Mutation Tests: Reference Range, Interpretation, Collection and Panels

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Life in the Lab: working in a malaria genome modification pipeline

Take on the role of a genome researcher and look at real cancer DNA datasets, taken from cancer patients, to find areas of mutation in the BRAF gene.

Phosphoinositide 3-kinase - Wikipedia, the free encyclopedia

Signal transduction - Akt/PKB signaling pathway - Wikipedia, the free encyclopedia

About half of all advanced melanomas have a defect in the BRAF gene.   A new strategy may extend the survival time of people living with the most serious form of skin cancer.

About half of all advanced melanomas have a defect in the BRAF gene. A new strategy may extend the survival time of people living with the most serious form of skin cancer.

Colorectal carcinoma is the most frequent type of bowel cancer and the second most common tumour disease among men and women in Germany. So-called microsatellite stable colorectal cancer with mutations in the BRAF gene represents a particularly aggressive form. The BRAF gene produces the enzyme B-Raf, ...

Colorectal carcinoma is the most frequent type of bowel cancer and the second most common tumour disease among men and women in Germany. So-called microsatellite stable colorectal cancer with mutations in the BRAF gene represents .

Clinical trial sends colorectal cancer patient into remission - WATCH THE VIDEO.    *** how to diagnose rectal cancer ***   Despite being diagnosed with chemotherapy-resistant colorectal cancer three years ago, Tim Givens is not only alive; he’s also in remission, thanks to a Phase I clinical trial. Testing revealed that Tim had a BRAF gene mutation, which keeps chemo from...

*** how to diagnose rectal cancer *** Despite being diagnosed with chemotherapy-resistant colorectal cancer three years ago, Tim Givens is not only alive; he’s also in remission, thanks to a Phase I clinical trial.

Vitamin C Cures Disease but Doctors and Pharmaceutical Companies Do Not Want You to Know This

The Swedish Society for Orthomolecular Medicine introduced Dr. Suzanne Humphries to lecture on the practical use of Vitamin C in Stockholm, Sweden.

What does LCH do?  78% have bone lesions, the most common area 50% have skin lesions 30% have lymph node involvement 20-50% have liver involvement • looks like primary sclerosing cholangitis & can be misdiagnosed as such • 75% need transplant -10% have Neurodegeneration -2% have lung involvement • looks like pulmonary fibrosis  20-30% have recurrences (relapse)  50-60% have the BRAF gene mutation • suspected there are additional (as yet unidentified) mutations responsible for the remainder

What does LCH do? 78% have bone lesions, the most common area 50% have skin lesions 30% have lymph node involvement 20-50% have liver involvement • looks like primary sclerosing cholangitis & can be misdiagnosed as such • 75% need transplant -10% have Neurodegeneration -2% have lung involvement • looks like pulmonary fibrosis 20-30% have recurrences (relapse) 50-60% have the BRAF gene mutation • suspected there are additional (as yet unidentified) mutations responsible for the remainder

A BRAF gene mutation in melanoma patients has been found to respond to MEK inhibitor drugs, providing a reason for routine screening and therapy in melanoma patients with the BRAF L597 mutation.

A BRAF gene mutation in melanoma patients has been found to respond to MEK inhibitor drugs, providing a reason for routine screening and therapy in melanoma patients with the BRAF mutation.

June 14, 2014 successful Phase III study results were shared from their melanoma trial. This comprehensive trial involved Cobimetinib and Zelboraf for treating patients with advanced cases of melanoma. All 495 patients had specific mutations of the BRAF gene and many were considered to have unresectable cases of this deadly skin cancer. The surprise came when they found these two drugs led to a longer progression free survival for patients in comparison to their treatment with Zelboraf…

June 14, 2014 successful Phase III study results were shared from their melanoma trial. This comprehensive trial involved Cobimetinib and Zelboraf for treating patients with advanced cases of melanoma. All 495 patients had specific mutations of the BRAF gene and many were considered to have unresectable cases of this deadly skin cancer. The surprise came when they found these two drugs led to a longer progression free survival for patients in comparison to their treatment with Zelboraf…

PET scan of a patient with metastatic melanoma, before & after treatment with a BRAF inhibitor

NY Times covers the heartbreak that is Melanoma..and a miracle drug

The New York Times is publishing a series on cancer research and a miracle drug for melanoma. These pics are before and after scans of a melanoma patient only 15 days after taking – a…

Fig. 1.

The BRAF gene has been found to be activated by mutation in human cancers, predominantly in malignant melanoma.

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